Nuclear Cardiology – PET

You can learn : 1) how to review and report PET for Inflammation/Sarcoidosis 2) how to interpret PET images vs ECG, Echo and MRI (when available)
     

1. Define what is physiologic testing for anomalous coronary arteries? 2. What stress test is best to create mechanical compression? 3. Use of treadmill PET and Dobutamine PET for risk stratification 4. Disadvantage of vasodilator PET
     

Challenges with prior CABG 1. Anatomic “landmarks” of various coronary arteries are not applicable 2. Segmental defects that are not contiguous 3. ECG changes that are not specific for ischemia 4. Abnormal septal motion 5. Flows and flow reserves are not well validated 6 Correlation with angiographic results may be challenging
     

Avoid the occulo-stenotic reflex. Review other imaging data if available. Severely fixed defects can be scar or hibernation. Unwise to open a coronary vessel to dead myocardium. Differentiate between LAD and diagonal/ramus.
     

Explain why we assess for hibernating myocardium, instances when assessment for hibernating myocardium is not indicated, and finally patient preparation and mini-tutorial on image display and reporting of findings.
     

We will guide you on the process we use for reading cardiac perfusion and metabolic PETs to evaluate myocardial hibernation. This process will allow you to identify myocardium at risk even when rest and stress perfusion images show a fixed perfusion defect.
     

Step by step review of identifying ischemia on cardiac PET and recognizing high risk markers.
     

This tutorial will walk you step by step through an abnormal PET study and help you identify high risk markers.
     

Identify areas of ischemia, correlate the ischemia to a coronary/ies territory, recognize TID and assign the scan a risk score.
     

Step by Step interpreting and reporting a normal cardiac PET scan.
     

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